Research Interest

Fluid and electrolyte physiology; interaction between osmolality and the renin-angiotensin- aldosterone axis; effects of pH and osmolality on glomerulosa cell function; evaluation of aldosterone secretion by means of factors not normally thought to inflence aldosterone secretion. Employ whole-animal models, isolated perfused adrenal glands, and primary cultures of glomerulosa cells.

Selected Publications

Books/chapters = 2. Journal articles = 64. Abstracts = 81. Video-tape and slide-tape teaching programs = 2.

1. Schneider, E. G. In water deprivation, osmolality becomes an important determinant of aldosterone secretion. News In International Physiological Sciences (NIPS), 5: 197-201, 1990.
2. Wang, W., N. Hayama, T. V. Robinson, R. E. Kramer, and E. G. Schneider. Effect of osmolality on cytosolic free calcium and aldosterone secretion. Am. J. Physiol., 262: E68-E75, 1992.
3. Hayama, N, W. Wang, T. V. Robinson, R. E. Kramer, and E. G. Schneider. Osmolality and potassium cause alterations in the volume of glomerulosa cells. Endocrinology 132:1230-1234, 1993.
4. Gosmanov, A.R., E.G. Schneider, and D.B. Thomason. NKCC activity restores muscle water during hyperosmotic challenge independent of insulin, ERK, and p38 MAPK. Am. J. Physiol. Regulatory Integrative Comp. Physiol. 285: R655-665, 2003.
5. Gosmanov, A.R., Z. Fan, X. Mi, E.G. Schneider, and D.B. Thomason. ATP-sensitive potassium channels mediate hyperosmotic stimulation of NKCC in slow-twitch muscle. Am. J. Physiol. Cell Physiol. 286: C586-C595, 2004.